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We've got to get a bit "science-y" to unravel some of this, but don't worry, I'm keeping it clear and focused on what's most important for you

GIP receptor activation complements these effects by further enhancing insulin secretion and may have effects on lipid metabolism and energy expenditure, though the latter mechanisms are still being characterized in humans

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It also promotes angiogenesis, or the formation of new blood vessels, which helps deliver nutrients and oxygen to damaged tissues

During fasting with decreased intra-portal insulin levels, hepatic GH sensitivity decreases leading to decreased serum IGF-I levels despite a compensatory increase in GH secretion

In acute healing estrogen acts to dampen the inflammatory response and in human studies prevents excessive neutrophil recruitment via downregulation of L-selectin, inhibiting homing to the wound site [172]